Innovative Research Award
Amira M. Badr — King Saud University, Saudi Arabia
Amira M. Badr’s documented publication activity covers pharmacology, toxicology, nephrotoxicity, and protective therapeutic mechanisms. Her ORCID record lists multiple peer-review activities, providing a documented basis for evaluating research contributions and scholarly engagement. [2]
Abstract
Amira M. Badr is a pharmacy and toxicology researcher, whose documented work addresses experimental mechanisms of organ injury and potential protective interventions. Her publication record includes studies of cisplatin-induced nephrotoxicity, oxidative stress, inflammatory signaling, and related pharmacological pathways. [3]
Keywords
Nephrotoxicity, Pharmacology, Toxicology, Oxidative Stress, Inflammation, Renal Injury, Pharmacological Protection, Molecular Signaling, Experimental Research, Drug Toxicity.
Introduction
The research profile documented for Amira M. Badr demonstrates sustained involvement in pharmacological and toxicological research. Her works examine mechanisms associated with tissue injury and investigate biological pathways through which candidate interventions may modify experimentally induced toxicity. [4]
Research Profile
Amira M. Badr’s research profile focuses on pharmacology and toxicology, with particular emphasis on nephrotoxicity, organ protection, and molecular mechanisms of drug- and chemical-induced injury. Her research also encompasses natural compounds, antioxidants, oxidative stress, nanocarriers, and emerging therapeutic strategies. [3]
Research Contributions
Her documented contributions include research on cisplatin-associated nephrotoxicity, renal injury, oxidative stress, and pharmacological protection. Representative studies investigate thymol, antioxidants, and molecular mechanisms associated with toxic injury and tissue protection. [3]
Publications
Selected documented publications include “Cuproptosis Contributes to Cisplatin-Induced Nephrotoxicity,” published in Pharmaceuticals in 2025, and “The Protective Effects of Vitamin B Complex on Diclofenac Sodium-Induced Nephrotoxicity,” published in Inflammation in 2024. [3]
Research Impact
The researcher profile reports 54 documents, 1,371 citations, and an h-index of 20. These indicators provide quantitative context for the researcher’s scholarly visibility, while the documented publication and review activity provides additional evidence of continuing engagement with academic research. [2]
Award Suitability
For the Innovative Research Award, the documented combination of pharmacology and toxicology research, sustained publication activity, and focused investigation of nephrotoxicity provides relevant evidence for consideration. Award assessment should remain based on verified scholarly records and the criteria established by Scientific World Research Awards. [5]
Conclusion
Amira M. Badr’s documented academic profile reflects research activity centered on pharmacology, toxicology, nephrotoxicity, oxidative stress, and related molecular mechanisms. Her indexed publications, researcher identifiers, citation indicators, and peer-review activity collectively provide a structured scholarly record relevant to academic recognition. [1]
External Links
References
- Elsevier. (n.d.). Scopus Author Details: Amira M. Badr, Author ID 57208267192. Scopus.
https://www.scopus.com/authid/detail.uri?authorId=57208267192
- ORCID. Works and peer-review activity recorded for Amira M. Badr.
https://orcid.org/0000-0003-3983-868X
- Al-Kharashi, L. A., Badr, A. M., et al. Cuproptosis Contributes to Cisplatin-Induced Nephrotoxicity: Insights into Thymol’s Potential Inhibitory and Protective Effects. Pharmaceuticals, 2025.
https://doi.org/10.3390/ph18111686
- Attia, H., Badr, A., et al. The Protective Effects of Vitamin B Complex on Diclofenac Sodium-Induced Nephrotoxicity: The Role of NOX4/RhoA/ROCK. Inflammation, 2024.
https://doi.org/10.1007/s10753-024-01996-6
- Badr, A. M., et al. Thymol Preserves Spermatogenesis and Androgen Production in Cisplatin-Induced Testicular Toxicity. Biomolecules, 2025.
https://doi.org/10.3390/biom15091277